Heart Rate Variability

The following graphs show the change in heart rate variability (HRV) before and after a crystal bowls sound healing session. Thank you to my client S L. (date of birth 20/5/74 ). This was done at Ubuntu wellness in Cape Town,  on 2/3/2011, using Heartmath equipment, challenge level 2  See Heartmath

Before crystal bowls therapy  at 7.16 pm



After crystal bowls therapy at 8.53 pm.


Heart rate variability (HRV) is a relatively new method for assessing the effects of stress on your body. It is measured as the time gap between your heart beats.   Heart rate variability (HRV) is the variation of beat to beat intervals, also known as R-R intervals. Electrocardiogram (ECG) is the electric signal originating from heart. The most distinct feature of the ECG is the QRS complex, which consists of the Q, R and S waves and originates from the electrical activation of the heart ventricles.

Figure 1 illustrates the variation of time between R-R intervals.


HRV indicates the fluctuations of heart rate; an average heart rate of 60 beats per minute (bpm) does not mean that the interval between successive heartbeats would be exactly 1.0 sec, instead they may fluctuate/vary from 0.5 sec up to 2.0 sec.

HRV is affected by aerobic fitness. HRV of a well-conditioned heart is generally high at rest. Other factors that affect HRV are age, genetics, body position, time of day, and health status. During exercise, HRV decreases as heart rate and exercise intensity increase. HRV also decreases during periods of mental stress and ill health. HRV is regulated by the autonomic nervous system. Parasympathetic activity decreases heart rate and increases HRV, whereas sympathetic activity increases heart rate and decreases HRV.

The importance of Heart Rate Variability for Your Health

We now know that the normal resting rhythm of the heart is highly variable rather than being monotonously regular, which was the widespread notion for many years
Heart rate variability, the change in the time intervals between adjacent heartbeats, is directly related to the body’s interdependent regulatory systems and ultimately, their efficiency and health. An optimal level of HRV within an organism reflects healthy function and an inherent self-regulatory capacity, adaptability, or resilience.
Generally the greater the HRV, the better, (although too much variability, or instability “such as arrhythmias or nervous system chaos is detrimental to health…) Too little variation indicates age-related system depletion, chronic stress, pathology, or inadequate functioning in various levels of self-regulatory control systems.


Short-term HRV analysis and assessment of the autonomic regulation

It is thought by many scientists, that Heart Rate Variability (HRV) will become as common as pulse, blood pressure or temperature in patient charts in the near future. In the last ten years more than 2000 published articles have been written about HRV. HRV has been used as a screening tool in many disease processes. . In diabetes and heart disease it has been proven to be predictive of the likelihood of future events.

Physiological Basics of HRV

The origin of heartbeat is located in a sino-atrial (SA) node of the heart, where a group of specialized cells continuously generates an electrical impulse spreading all over the heart muscle through specialized pathways and creating process of heart muscle contraction well synchronized between both atriums and ventricles. The SA node generates such impulses about 100-120 times per minute at rest. However in healthy individual resting heart rate (HR) would never be that high. This is due to continuous control of the autonomic nervous system (ANS) over the output of SA node activity, which net regulatory effect gives real HR. In healthy subject at rest it is ranging between 50 and 70 beats per minute.

Schematic explanation of RA, LA, RV, LV parameters and their visualisation on Heart Rate


Autonomic nervous system. The autonomic nervous system is a part of the nervous system that non-voluntarily controls all organs and systems of the body. As the other part of nervous system ANS has its central (nuclei located in brain stem) and peripheral components (afferent and efferent fibers and peripheral ganglia) accessing all internal organs. There are two branches of the autonomic nervous system – sympathetic and parasympathetic (vagal) nervous systems that always work as antagonists in their effect on target organs.

Sympathetic nervous system. For most organs including heart the sympathetic nervous system stimulates organ’s functioning. An increase in sympathetic stimulation causes increase in HR, stroke volume, systemic vasoconstriction, etc. The heart response time to sympathetic stimulation is relatively slow. It takes about 5 seconds to increase HR after actual onset of sympathetic stimulation and almost 30 seconds to reach its peak steady level.

Schema explaining how parasympathetic and sympathetic nervous systems inhibit functioning organs.


Parasympathetic nervous system. In contrast, the parasympathetic nervous system inhibits functioning of those organs. An increase in parasympathetic stimulation causes decrease in HR, stroke volume, systemic vasodilatation, etc. The heart response time to parasympathetic stimulation is almost instantaneous. Depending on actual phase of heart cycle it takes just 1 or 2 heartbeats before heart slows down to its minimum proportional to the level of stimulation.At rest both sympathetic and parasympathetic systems are active with parasympathetic dominance. The actual balance between them is constantly changing in attempt to achieve optimum considering all internal and external stimuli.
There are various factors affecting autonomic regulation of the heart, including but not limited to respiration, thermoregulation, humoral regulation (rennin-angiotensin system), blood pressure, cardiac output, etc. One of the most important factors is blood pressure. There are special baroreceptive cells in the hear and large blood vessels that sense blood pressure level and send afferent stimulation to central structures of the ANS that control HR and blood vessel tonus primarily through sympathetic and somewhat parasympathetic systems forming continuous feedback dedicated to maintain systemic blood pressure. This mechanism is also called baroreflex, which increases HR when blood pressure decreases and vice versa. This mechanism is also targeted to maintain optimal cardiac output.

Schema showing the baroreflex functionality


Heart Rate Variability in research

The HRV analysis is a powerful, very accurate, reliable, reproducible, yet simple to do. It is found that lowered HRV is associated with aging, decreased autonomic activity, hormonal tonus, unspecific types of autonomic neuropathies (e.g. diabetic neuropathy) and increased risk of sudden cardiac death after acute heart attack. Other research indicated that depression, panic disorders and anxiety have negative impact on autonomic function, typically causing depletion of the parasympathetic tonus. On the other hand an increased sympathetic tonus is associated with lowered threshold of ventricular fibrillation. These two factors could explain why such autonomic imbalance caused by significant mental and emotional stress increases risk of heart attack followed by sudden cardiac death.
Aside from that, there are multiple studies indicating that HRV is quite useful as a way to quantitatively measure physiological changes caused by various interventions both pharmacological and non-pharmacological during treatment of many pathological conditions having significant manifestation of lowered HRV.
However it is important to realize that clinical implication of HRV analysis has been clearly recognized in only two medical conditions:
1. Predictor of risk of arrhythmic events or sudden cardiac death after acute heart attack
2. Clinical marker of diabetic neuropathy evolution
Nevertheless, as the number of clinical studies involving HRV in various clinical aspects and conditions grows, HRV remains one of the most promising methods of investigating general health in the future